Publications

Protein 4.1N Plays a Cell Type-Specific Role in Hippocampal Glutamatergic Synapse Regulation

Schizophrenia-associated SAP97 mutations increase glutamatergic synapse strength in the dentate gyrus and impair contextual episodic memory in rats.

Autism Spectrum Disorder/Intellectual Disability-Associated Mutations in Trio Disrupt Neuroligin 1-Mediated Synaptogenesis.

An optogenetic method for investigating presynaptic molecular regulation

Kalirin and Trio: RhoGEFs in Synaptic Transmission, Plasticity, and Complex Brain Disorders

Synaptic Kalirin-7 and Trio Interactomes Reveal a GEF Protein-Dependent Neuroligin-1 Mechanism of Action.

Tiam1 is Critical for Glutamatergic Synapse Structure and Function in the Hippocampus.

An Intellectual Disability-Related Missense Mutation in Rac1 Prevents LTP Induction.

An autism spectrum disorder-related de novo mutation hotspot discovered in the GEF1 domain of Trio