Lorraine Turcotte

• Post-Doctoral Fellow, Clinical Research Institute, Montreal, Canada, 08/01/1991-06/30/1993
• Post-Doctoral Fellow, August Krogh Institute, University of Copenhagen, 08/01/1988-07/01/1991

Tenure Track Appointments

• Professor of Biological Sciences, University of Southern California, 2013 –
• Chair, Department of Kinesiology, University of Southern California, 08/15/2004 – 08/15/2010
• Associate Professor, Department of Biological Sciences, University of Southern California, 01/01/2003 – 08/15/2013
• Associate Professor, Department of Kinesiology, University of Southern California, 01/01/1999 – 08/15/2010
• Director, Metabolic Regulation Lab, University of Southern California, 01/01/1994 –
• Assistant Professor, University of Southern California, 01/01/1993 – 01/01/1999

PostDoctoral Appointments

• Post-Doctoral Fellow, Hotel Dieu Hospital, 07/1991 – 07/1993
• Post-Doctoral Fellow, University of Copenhagen, 08/1988 – 07/1991

Other Employment

• Member-at-Large, Academic Senate, 07/01/2023-06/30/2024
• Section Head, Human and Evolutionary Biology, University of Southern California, 2019
• Gabilan Distinguished Professor of Science and Engineering, University of Southern California, 08/2017-07/2020

Summary Statement of Research Interests

Our overall research goal is to better understand the signaling factors that regulate metabolism in health and disease and how to improve human performance. We are specifically interested in unraveling the role of signaling molecules and mitochondrial dysfunction in the development of metabolic pathologies such as obesity and type II diabetes. We are also interested in investigating the role played by inflammation in human performance deficits. Our research projects include studying (1) how genetics alter mitochondrial function and exercise performance, and (2) how hormonal regulation controls metabolic function and muscle performance in cold water immersion.

Contracts and Grants Awarded

• HAART protease inhibitor-induced insulin resistance in skeletal muscle, (American College of Sports Medicine), Lorraine P Turcotte, Lindsey D Bogachus, $5,000, 07/01/2010 – 06/30/2011
• Connecting physiological systems and human movement using multimedia tools, (National Science Foundation), Lorraine Turcotte and Jill McNitt-Gray, $119,163, 08/01/2000 – 06/30/2004
• Exercise and skeletal muscle fatty acid metabolism, (National Institutes of Health), Lorraine Turcotte, $350,000, 07/01/1998 – 06/30/2003

USC Funding

• WiSE Programs. Antiretroviral therapy and skeletal muscle insulin resistance: IR induced by antiretroviral therapy is due to decreased insulin signaling and is mediated by a decrease in FA oxidative capacity and an increase in the activation of nuclear factor kappaB., $25000, 07/2009 – 06/2011
• Undergraduate Reseach Associates Program. The role of AMP-activated protein kinase in the prevention of obesity and type II diabetes: The role of AMPK in the development of obesity and type II diabetes will be tested in transgenic that do not express AMPK in muscle. , $10000, 2008-2009
• WiSE Faculty Research Award. Regulation of muscle fatty acid metabolism: To collect data on the effects of downregulation of AMPKalpha2 on the regulation of fatty acid metabolism during muscle contractions and during high fat feeding. , $21533, 07/01/2004 – 06/30/2006
• Innovative Undergraduate Teaching, Center for Excellence in Teaching. Using multimedia tools in laboratory sessions to enhance the learning of muscle physiologic concepts and to promote the understanding of the integration of the concepts with mechanical principles: CD-ROM of metabolic pathways was completed. , $9138, 2002-2003
• Zumberge Interdisciplinary Research Grant. Fatty acid metabolism and intracellular signaling: Gene expression of SIRT1 and RIP140 was genetically silenced in L6 cells. Results showed that both intracellular cascades have important roles in the regulation of metabolism. , $43000, 07/01/2001 – 06/30/2003
• WiSE Faculty Research Award. Fatty acid metabolism in muscle cells: We showed that AMP-activated protein kinase is critical to the regulation of muscle metabolism. , $24600, 07/01/2001 – 06/30/2003
• Undergraduate Research Associated Program. Effects of aging on muscle fatty acid metabolism: Effects of aging on the content of proteins and transporters implicated in the regulation of fatty acid metabolism were investigated in the Fischer 344/Brown Norway rat model. , $6900, 2000-2001
• Innovative Undergraduate Teaching, Center for Excellence in Teaching. Using multimedia techniques in laboratory sessions to enhance learning of the metabolic pathways in lecture: Development of incubated muscle preparation in undergraduate physiology labs., $6000, 2000-2001

Conference Presentations

• AMPKa2 down-regulation prevents voluntary wheel running-induced changes in glucose uptake but not in fatty acid uptake in mouse skeletal muscle , American College of Sports MedicinePoster, American College of Sports Medicine, San Francisco, Spring 2012
• Low RIP140 expression rescues basal FA uptake via differential expression of FATP1 and CD36 in skeletal muscle cells exposed to high palmitate , Experimental BiologyPoster, American Physiological Society, San Diego, Spring 2012
• Low RIP140 expression uncovers the central role of the AKT-PKCzeta axis in the regulation of insulin-mediated fatty acid oxidation in skeletal muscle cells , American College of Sports MedicinePoster, American College of Sports Medicine, San Francisco, Spring 2012
• Under insulin-mediated conditions, resveratrol restores glucose uptake but not fatty acid oxidation in skeletal muscle cells made insulin resistant by treatment with protease inhibitors , American College of Sports MedicinePoster, American College of Sports Medicine, San Francisco, Spring 2012
• Calcium and Metabolic Regulation , 5th Asia-Pacific Conference on Exercise and Sports ScienceTalk/Oral Presentation, Chinese Ministry, Invited, Shanghai, China, Fall 2011
• Atazanavir sulfate induces insulin resistance in part via an increase in JUNK1/2 phosphorylation in skeletal muscle cells , American College of Sports MedicinePoster, American College of Sports Medicine, Denver, Spring 2011
• Contraction-induced signaling: Evidence of convergent cascades in the regulation of muscle metabolism , 4th Basel Muscle SymposiumTalk/Oral Presentation, Swiss Medical Research Council, Invited, Basel, Switzerland, Spring 2011
• Low Receptor Interacting Protein 140 (RIP140) expression adversely impacts proximal insulin signaling in L6 skeletal muscle cells , Experimental BiologyPoster, American Physiological Society, Washington, DC, Spring 2011
• Metformin blunts highly active antiretroviral therapy-induced insulin resistance in skeletal muscle cells , American College of Sports MedicineTalk/Oral Presentation, American College of Sports Medicine, Denver, Spring 2011
• AMPK-SIRT1 signaling: Can drinking red wine really help in preventing insulin resistance? , Southwest American College of Sports MedicineTalk/Oral Presentation, Southwest American College of Sports Medicine, Invited, San Diego, Fall 2010
• AMP-activated protein kinase provides evidence for a Metformin-induced decrease in FA uptake and oxidation and increase in glucose uptake and SIRT1 activity in L6 skeletal muscle cells , Experimental BiologyPoster, American Physiological Society, Anaheim, Spring 2010
• AMPKa2 deletion leads to marked alterations in SIRT1 activity, CPT1 expression and basal FA muscle metabolism in high-fat-fed mice , Experimental BiologyPoster, American Physiological Society, Anaheim, Spring 2010
• AMPKa2 is not necessary to increase the activation state of ERK1/2, CaMKI, or AS160 during moderate intensity muscle contraction in perfused mouse muscle , Experimental BiologyPoster, American Physiological Society, Anaheim, Spring 2010
• Low RIP140 expression partially restores metabolic flexibility in L6 muscle cells treated with high FA , Annual Biology Interdisciplinary Graduate Student SymposiumPoster, Biology Interdepartmental Graduate Student Associa, Invited, USC, Spring 2010
• Low RIP140 expression partially restores metabolic flexibility in L6 muscle cells treated with high FA , Experimental BiologyPoster, American Physiological Society, Anaheim, Spring 2010
• Receptor Interacting Protein 140 (RIP140) silencing via RNAi interferes with fatty acid metabolism but not with glucose uptake in L6 cells , RNAi & miRNA World CongressPoster, RNAi miRNA World Congress, Boston, Spring 2010
• AICAR and Metformin preferentially activate different AMP-activated protein kinase (AMPK) isozymes to regulate muscle metabolism and SIRT1 activity in L6 muscle cells , SouthWest American College of Sports MedicineTalk/Oral Presentation, SouthWest American College of Sports Medicine, Invited, San Diego, Fall 2009
• Deletion of muscle AMPKa2 abrogates high fat diet-induced IL6 expression in adipose tissue , SouthWest American College of Sports MedicinePoster, SouthWest American College of Sports Medicine, San Diego, Fall 2009
• Suppression of receptor interacting protein (RIP140) expression in L6 cells impairs the regulation of insulin-sensitive FA uptake and oxidation but not that of insulin-sensitive glucose uptake , USC Graduate and Professional Student Appreciation Week Research FairPoster, USC, Los Angeles, Fall 2009
• Suppression of Receptor Interacting Protein 140 (RIP140) expression in L6 cells impairs the regulation of insulin-sensitive FA uptake and oxidation but not that of insulin-sensitive glucose uptake , SouthWest American College of Sports MedicinePoster, SouthWest American College of Sports Medicine, San Diego, Fall 2009
• AICAR and Metformin increase SIRT1 activity despite preferentially phosphorylating different AMPK isoforms , International Biochemistry of ExerciseTalk/Oral Presentation, International Biochemistry of Exercise, Invited, Guelph, Ontario, Spring 2009
• AMPKa2 is not necessary in the regulation of Ca2+-induced FA metabolism in perfused mouse muscle , Internatioal Biochemistry of ExercisePoster, Internatioal Biochemistry of Exercise, Guelph, Ontario, Spring 2009
• AMPKa2 is not necessary in the regulation of Ca2+-induced FA metabolism in perfused mouse muscle , USC Biology Interdepartmental Graduate SymposiumTalk/Oral Presentation, USC Biology Interdepartmental Graduate Student Ass, Invited, USC, Spring 2009
• Contrary to AICAR, Metformin regulates fatty acid metabolism by preferentially phosphorylating AMPKa1 in L6 myotubes , USC Graduate and Professional Student Appreciation Week Research Fair Poster, Graduate & Professional Student Association, USC, Spring 2009
• Fatty acid metabolism and glucose uptake are differently affected by aspirin treatment in muscle cells exposed to high saturated fatty acid availability , USC Graduate and Professional Student Appreciation Week Research Fair Poster, Graduate & Professional Student Association, USC, Spring 2009
• Key role for AMPKa2 in the regulation of contraction-mediated fatty acid metabolism in perfused mouse muscle , American College of Sports MedicineTalk/Oral Presentation, American College of Sports Medicine, Invited, Seattle, Spring 2009
• Selective AMPKa1 and AMPKa2 deficiency uncovers isozyme-specific regulation of metabolism and SIRT1 activation in L6 muscle cells , American College of Sports MedicinePoster, American College of Sports Medicine, Seattle, Spring 2009
• Selective AMPKa1 and AMPKa2 deficiency uncovers isozyme-specific regulation of metabolism and SIRT1 activation in L6 muscle cells , USC Biology Interdepartmental Graduate SymposiumPoster, USC Biology Interdepartmental Graduate Student Ass, USC, Spring 2009
• Evidence for the involvement of CaMKKbeta in the regulation of glucose uptake in perfused rat muscle , Biology of ExercisePoster, American Physiological Society, Hilton Head, Fall 2008
• Contraction-induced fatty acid uptake and oxidation are regulated in part via CaMKKbeta-dependent AMPK activation in perfused rat muscle , Experimental BiologyPoster, American Physiological Society, San Diego, Spring 2008
• Calcium/calmodulin-dependent protein kinase regulates fatty acid uptake and oxidation during moderate intensity muscle contraction in part via activation of AMP-activated protein kinase , Southwest American College of Sports MedicineTalk/Oral Presentation, American College of Sports Medicine, Invited, San Diego, Fall 2007
• Acute and chronic elevations in intracellular calcium increases FA uptake an oxidation via different molecular mechanisms in L6 myotubes , Medicine and Science in Sports and ExerciseTalk/Oral Presentation, American College of Sports Medicine, Invited, New Orleans, Spring 2007
• Chronic activation of AMPK enhances insulin-sensitive fatty acid uptake but not oxidation in L6 cells , Experimental BiologyTalk/Oral Presentation, American Physiological Society, Invited, San Francisco, Spring 2006
• Cross-talk between CaMKII and AMPK is associated with increased FA metabolism in contracting rodent muscle , Experimental BiologyPoster, American Physiological Society, San Francisco, Spring 2006
• ERK1/2 inhibition prevents the contraction-induced increase in FA metabolism and plasma membrane CD36 content, without affecting AMPKalpha2 activity, in a dose-dependent manner in rodent muscle , Southwest American College of Sports MedicineTalk/Oral Presentation, American College of Sports Medicine, Invited, Las Vegas, Fall 2005
• aPKC-zeta inhibition abolishes insulin-induced LCFA uptake but does not prevent the insulin-induced decreased in LCFA oxidation , Medicine and Science in Sports and ExerciseTalk/Oral Presentation, American College of Sports Medicine, Nashville, Spring 2005
• Contraction-induced ERK1/2 activation is not involved in the regulation of muscle FA oxidation during contraction of increasing intensity , Experimental BiologyPoster, American Physiological Society, San Diego, Spring 2005
• Inhibition of PI3K but not PKB/Akt prevents the insulin-induced effects on LCFA metabolism , Experimental BiologyPoster, American Physiological Society, San Diego, Spring 2005
• Regulation of contraction-induced FA uptake by AMPK and ERK1/2 is intensity-dependent in rodent muscle , Medicine and Science in Sports and ExerciseTalk/Oral Presentation, American College of Sports Medicine, Invited, Nashville, Spring 2005

Book Chapters

• Turcotte, L. P. (2006). Lipid metabolism during exercise. pp. p.105-136. Champaign, Illinois: Exercise Metabolism/Human Kinetics.
• Turcotte, L. P. (1999). Fatty acid binding proteins and lipid metabolism in skeletal muscle. pp. 201-215. Champaign, Illinois: Human Kinetics Publishers.

Journal Article

• Jensen, A. E., Neiderberger, B., Jaworski, R., Devaney, J. M., Turcotte, L. P., Kelly, K. R. (2019). Tumor Necrosis Factor alpha stress response is reduced following load carriage training. Military Medicine. Vol. 184 (1/2), pp. e256-e260.
• Moore, T. M., Zhou, Z., Cohn, W., Nordheim, F., Lin, A. J., Kalajian, N., Strumwasser, A. R., Corey, K., Whitney, K., Ho, T., Ho, T., Lee, J. L., Rucker, D. H., Shirahi, O., van der Bliek, A. M., Whitelegge, J. P., Seldin, M. M., Lusis, A. J., Lee, S., Drevon, C. A., Mahata, S. K., Turcotte, L. P., Hevener, A. L. (2019). The impact of exercise on mitochondrial dynamics and the role of Drp1 in exercise performance and training adaptations in skeletal muscle. Molecular Metabolism. Vol. 21, pp. 51-67.
• Conoscenti, M. A., Williams, N. M., Turcotte, L. P., Minor, T. R., Fanselow, M. S. (2019). Post-stress fructose and glucose ingestion exhibit dissociable behavioral and physiological effects. Nutrients. Vol. 11 (2), pp. 361.
• Jensen, A. E., Palombo, L. J., Niederberger, B., Turcotte, L. P., Kelly, K. R. (2016). Exercise training with blood flow restriction has little effect on muscular strength and does not change IGF-1 in fit military warfighters. Growth Hormone and IGF Research. Vol. 27, pp. 33-40.
• Abbott, M. J., Constantinescu, S., Turcotte, L. P. (2012). AMPKa2 is an essential signal in the regulation of insulin-stimulated fatty acid uptake in control-fed and high fat-fed mice. Experimental Physiology. PubMed Web Address
• Kelly, K. R., Turcotte, L. P. (2012). AMP-regulated kinase and calcium/calmodulin-dependent kinase II co-regulate fatty acid uptake and oxidation in L6 muscle cells. Recent Research Development in Physiology. Vol. 5, pp. 15-26.
• Abbott, M. J., Bogachus, L. D., Turcotte, L. P. (2011). AMPKalpha2 deficiency uncovers time-dependency in the regulation of cotnraction-induced palmitate and glucose uptake in mouse muscle. Journal of Applied Physiology. Vol. 111, pp. 125-134.
• Ali, A. H., Koutsari, C., Mundi, M., Stegall, M. D., Heimbach, J. K., Taler, S. J., Nygren, J., Thorell, A., Bogachus, L. D., Turcotte, L. P., Bernlohr, D., Jensen, M. D. (2011). Free fatty acid storage in omental and abdominal subcutaneous adipose tissue – effects of sex, lipogenic enzyme activity and transport proteins. Diabetes. Vol. 60, pp. 2300-2307.
• Bogachus, L. D., Turcotte, L. P. (2011). HIV protease inhibitors induce metabolic dysfunction in part via increased JNK1/2 pro-inflammatory signaling in L6 cells. Antiviral Research. Vol. 92, pp. 415-423.
• Bogachus, L. D., Turcotte, L. P. (2010). Genetic downregulation of AMPKalpha1 and AMPKalpha2 uncovers the mechanism by which metformin decreases FA uptake and oxidation in skeletal muscle cells. America Journal of Physiology, Cell. Vol. 299, pp. C1549-C1561.
• Kelly, K. R., Abbott, M. J., Turcotte, L. P. (2010). Short-term AMPK activation enhances insulin-sensitive FA uptake and increases the effects of insulin on FA oxidation in L6 muscle cells. Exper Biol Med. Vol. 235, pp. 514-521.
• Abbott, M. J., Edelman, A. M., Turcotte, L. P. (2009). CaMKK is an upstream signal of AMP-activated protein kinase in regulation of substrate metabolism in contracting skeletal muscle. Am J Physiol Regul Integr Comp Physiol. Vol. 297, pp. R1724-R1732.
• Turcotte, L. P., Fisher, J. S. (2008). Skeletal muscle insulin resistance: Roles of fatty acid metabolism and exercise. Physical Therapy. Vol. 88 (11), pp. 1279-1296.
• Kelly, K. R., Sung, C. K., Abbott, M. J., Turcotte, L. P. (2008). Phosphatidylinositol 3-kinase-dependent insulin regulation of long-chain fatty acid (LCFA) metabolism in L6 cells: involvement of atypical protein kinase C-zeta in LCFA uptake but not oxidation. Journal of Endocrinology. Vol. 198, pp. 375-384.
• Raney, M. A., Turcotte, L. P. (2008). Evidence for the involvement of CaMKII and AMPK in Ca+2-dependent signaling pathways regulating FA uptake and oxidation in contracting rodent muscle. Journal of Applied Physiology. Vol. 104, pp. 1366-1373.
• Berthiaume, M., Laplante, M., Festuccia, W., Cianflore, K., Turcotte, L., Joanisse, D., Olivecrone, G., Deshaies, Y. (2007). 11-ß-HSD-1 inhibition improves triglyceridemia through reduced liver VLDL secretion and partitions lipids towards oxidative tissues. Am J Physiol Endocrinol Metab. Vol. 293, pp. E1054-E1052.
• Raney, M. A., Turcotte, L. P. (2007). Evidence for the regulation of contraction-induced fatty acid oxidation via extracellular signal-regulated kinase 1/2 activation independent of changes in fatty acid uptake. Metabolism. Vol. 56, pp. 1192-1200.
• Todd, M. K., Watt, M. J., Le, J., Hevener, A. L., Turcotte, L. P. (2007). Thiazolidinediones enhance skeletal muscle triacylglecerol synthesis while protecting against fatty acid-induced inflammation and insulin resistance. Am J Physio Endocrinol Metab. Vol. 292, pp. E485-E493.
• Raney, M. A., Turcotte, L. P. (2006). Regulation of contraction-induced FA uptake and oxidation by AMPK and ERK1/2 is intensity dependent in rodent muscle. Am J Physiol Endocrinol Metab. Vol. 291, pp. E1220-E1227.
• Raney, M. A., Yee, A. J., Todd, M. K., Turcotte, L. P. (2005). AMPK activation is not critical in the regulation of muscle FA uptake and oxidation during low-intensity muscle contraction. Am J Physiol Endocrinol Metab. Vol. 288, pp. E592-E598.
• Turcotte, L. P., Raney, M. A., Todd, M. K. (2005). ERK1/2 inhibition prevents contraction-induced increase in plasma membrane FAT/CD36 content and FA uptake in rodent muscle. Acta Physiol Scand. Vol. 184, pp. 131-139.
• Tucker, M. Z., Turcotte, L. P. (2005). Brief food restriction in old animals decreases TG content and insulin-stimulated TG synthesis. J Gerontol Biol Sci. Vol. 60A, pp. 157-164.
• Todd, M. K., Yaspelkis III, B. B., Turcotte, L. P. (2005). Short-term leptin treatment increases fatty acid uptake and oxidation in muscle of high fat-fed rats. Metabolism. Vol. 54, pp. 1218-1224.
• Tucker, M. Z., Turcotte, L. P. (2003). Aging is associated with elevated muscle triglyceride content and increased insulin-stimulated fatty acid uptake. Am J Physiol Endocrinol Metab. Vol. 283, pp. E827-E835.
• Turcotte, L. P. (2003). Mitochondria: Biogenesis, structure and function. Med Sci Sports & Exerc. Vol. 35, pp. 82-85.
• Tucker, M. Z., Turcotte, L. P. (2002). Impaired fatty acid oxidation in muscle of aging rats perfused under basal conditions. Am J Physiol Endocrinol Metab. Vol. 282, pp. E1102-E1109.
• Turcotte, L. P., Tucker, M. Z. (2002). Brief food restriction increases FA oxidation and glycogen synthesis under insulin-stimulated conditions. Am J Physiol Regulatory Integrative and Comp Physiol. Vol. 282, pp. R1210-R1218.
• Yee, A. J., Turcotte, L. P. (2002). Insulin fails to alter plasma LCFA metabolism in muscle perfused at similar glucose uptake. Am J Physiol Endocrinol Metab. Vol. 283, pp. E73-E77.
• Turcotte, L. P. (2002). High carbohydrate availabillity increases LCFA uptake and decreases LCFA oxidation in perfused muscle. Am J Physiol Endocrinol Metab. Vol. 282, pp. E177-E183.
• Luiken, J. F., Arumugan, Y., Dyck, D. J., Bell, R. C., Pelsers, M. M., Turcotte, L. P., Tandon, N. N., Glatz, J. C., Bonen, A. (2001). Increased rates of fatty acid uptake and plasmalemmal fatty acid transporters in obese Zucker rats. J Biol Chem. Vol. 276, pp. 40567-40573.
• Turcotte, L. P., Swenberger, J. R., Tucker, M. Z., Yee, A. J. (2001). Increased fatty acid uptake and altered fatty acid metabolism in insulin-resistant muscle of obese Zucker rats. Diabetes. Vol. 50, pp. 1389-1396.
• Turcotte, L. P. (2000). Muscle fatty acid uptake during exercise: Possible mechanisms. Exercise and Sport Sciences Reviews. Vol. 28, pp. 4-9.
• Turcotte, L. P., Swenberger, J. R., Tucker, M. Z., Yee, A. J., Trump, G., Luiken, J. F., Bonen, A. (2000). Muscle palmitate transport and binding are saturable and inhibited by antibodies to FABP (PM). Mol Cell Biochem. Vol. 210, pp. 53-63.
• Gazdag, A. C., Wetter, T. J., Davidson, R. T., Robinson, K. A., Buse, M. G., Yee, A. J., Turcotte, L. P., Carter, G. D. (2000). Lower calorie intake enhances muscle insulin action and reduces hexosamine levels. Am J Physiol Regulatory Integrative & Comp Physiol. Vol. 278, pp. R504-R512.
• Turcotte, L. P. (1999). Nutritional aspects of exercise. Role of fats in exercise: Types and quality. Clinics in Sports Medicine. Vol. 18, pp. 485-498.
• Simoneau, J., Veerkamp, J. H., Turcotte, L. P., Kelley, D. E. (1999). Metabolic markers of capacity to utilize fatty acids in human skeletal muscle: Relation to insulin resistance, obesity and weight loss. FASEB Journal. Vol. 13 (2051-2060)
• Luiken, J. J., Turcotte, L. P., Bonen, A. (1999). Protein-mediated palmitate uptake and expression of fatty acid transport proteins in heart giant vesicles. Journal of Lipid Research. Vol. 40, pp. 1007-1016.
• Turcotte, L. P., Swenberger, J. R., Tucker, M. Z., Yee, A. J. (1999). Training-induced elevation in FABPpm is associated with increased palmitate use in contracting muscle. Journal of Applied Physiology. Vol. 87, pp. 285-293.

• Recognition Award, SouthWest American College of Sports Medicine, Fall 2009
• Mellon Award for Excellence in Mentoring, University of Southern California, Spring 2008
• Remarkable Women Award, University of Southern California, 2004
• Innovative Teaching Award: USC Center for Excellence in Teaching Innovative Undergraduate Teaching Award, 2002-2003
• Faculty of the Month, Mortar Board National Honor Society, 2001
• Innovative Teaching Award: USC Center for Excellence in Teaching Innovative Undergraduate Teaching Award, 2000-2001
• Professor of the Year, Gamma Sigma Alpha Honor Society, 1999
• Fellow, American College of Sports Medicine, Fellow, Spring 1996

Administrative Appointments

• Section Head, 01/01/2019 – 08/15/2021
• Chair of the Department of Kinesiology, 08/15/2007 – 08/15/2010
• Chair of the Department of Kinesiology, 09/01/2004 – 08/15/2007

Administative Appointment

• President (Elect, President, Past), American College of Sports Medicine, Southwest Chapter, 2013 – 2016
• Chair, Conference Exhibit Committee, American College of Sports Medicine, Southwest Chapter, 07/01/2000 – 06/30/2003
• Chair, International Scholar Award Committee, International Relations Committee, American College of Sports Medicine, 07/01/1997 – 06/30/2003
• Presidential Task Force, American College of Sports Medicine, 07/01/2001 – 06/30/2002

Editorships and Editorial Boards

• Associate Editor, Canadian Journal of Physiology and Pharmacology, 2011 – 2017
• Review Editor, Frontiers in Fatty Acid and Lipid Physiology, 2011 – 2014
• Associate Editor, Exercise and Sport Sciences Reviews, 2002 – 2005
• Editorial Board, Health and Fitness Journal, 2001 – 2004

Professional Offices

• Trustee (National election), Board of Trustees, American College of Sports Medicine, 05/2009 – 06/2012
• Executive Council Member, American College of Sports Medicine, Southwest Chapter, 07/01/2000 – 06/30/2003

Professional Memberships

• American Physiological Society, Member, 1993 –
• American College of Sports Medicine, Fellow, 1982 –
• American Diabetes Association, Member Council on Exercise, 1988 – 2010
• International Society for the Study of Fatty Acids and Lipids, Member, 1995 – 2004